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Emerging role of interleukin-33 in autoimmune diseases
Pei, Cheng1,2; Barbour, Mark1; Fairlie-Clarke, Karen J.1; Allan, Debbie1; Mu, Rong3; Jiang, Hui-Rong1
关键词autoimmune diseases inflammation interleukin-33 ST2
刊名IMMUNOLOGY
2014
DOI10.1111/imm.12174
141期:1页:9-17
收录类别SCI
文章类型Review
WOS标题词Science & Technology
类目[WOS]Immunology
研究领域[WOS]Immunology
关键词[WOS]INFLAMMATORY-BOWEL-DISEASE ; MEDIATED AIRWAY INFLAMMATION ; INNATE LYMPHOID-CELLS ; ULCERATIVE-COLITIS ; MAST-CELLS ; INDUCED ARTHRITIS ; SOLUBLE ST2 ; RHEUMATOID-ARTHRITIS ; ATOPIC-DERMATITIS ; IL-33 EXPRESSION
英文摘要

Interleukin-33 (IL-33) is a member of the IL-1 cytokine family. It predominantly induces type 2 immune responses and thus is protective against atherosclerosis and nematode infections but contributes to allergic airway inflammation. Interleukin-33 also plays a pivotal role in the development of many autoimmune diseases through mechanisms that are still not fully understood. In this review, we focus on the recent advances in understanding of the expression and function of IL-33 in some autoimmune disorders, aiming to provide insight into its potential role in disease development.

语种英语
WOS记录号WOS:000327947000002
资助机构Medical Research Scotland
引用统计
被引频次:34[WOS]   [WOS记录]     [WOS相关记录]
文献类型期刊论文
条目标识符http://ir.bjmu.edu.cn/handle/400002259/63137
专题北京大学第二临床医学院
北京大学第二临床医学院_风湿免疫科
作者单位1.Univ Strathclyde, Strathclyde Inst Pharm & Biomed Sci, Glasgow G4 0RE, Lanark, Scotland
2.Xi An Jiao Tong Univ, Coll Med, Affiliated Hosp 1, Dept Ophthalmol, Xian 710049, Peoples R China
3.Peking Univ, Peoples Hosp, Inst Rheumatol & Immunol, Beijing 100871, Peoples R China
推荐引用方式
GB/T 7714
Pei, Cheng,Barbour, Mark,Fairlie-Clarke, Karen J.,et al. Emerging role of interleukin-33 in autoimmune diseases[J]. IMMUNOLOGY,2014,141(1):9-17.
APA Pei, Cheng,Barbour, Mark,Fairlie-Clarke, Karen J.,Allan, Debbie,Mu, Rong,&Jiang, Hui-Rong.(2014).Emerging role of interleukin-33 in autoimmune diseases.IMMUNOLOGY,141(1),9-17.
MLA Pei, Cheng,et al."Emerging role of interleukin-33 in autoimmune diseases".IMMUNOLOGY 141.1(2014):9-17.
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